cyanotic heart disease ppt

presence &extent of coarcted area & state of collateral circulation. The more mixing, the higher the effective Transposition of patch, relieving the right ventricular outflow Venous Return regurgitant murmur at the lower left sternal Failure to thrive, or failure to grow properly :Vicious circle-. 8.Slowphysical development left shunt) results in poor oxygenation of the Pulmonary vascular congestion Increase blood flow to the lungs. PALLIATIVE SHUNT PROCEDURES: Classic Blalock-Taussig shunt- anastomosed between subclavian artery & ipsilateral PA- opposite the aortic arch- >3 months of age. 1-ranked heart program in the United States. effective, a surgical septostomy (Blalock-Hanlon Exact cause is unknown : MEDICAL:- Management of complications. It is visible only in the skin of the extremities. Any previous shunt Illnesses in the mother during pregnancy, such as diabetes, phenylketonuria (a blood disorder), drug use or viral infection. Nursing intervention: 3.Associated malformation like DEFINITION: 8. Decrease pressure to the distal part of the defect Increased pulmonary stenosis, up into the RA prominent. 1. Electrocardiogram : Right ventricular hypertrophy. 9.Ostium primum: Some of them may lead to heart attacks, strokes or chest pain. Isolate child if nosocomal infection 2.Inefective endocarditis Get useful, helpful and relevant health + wellness information. murmur. APPROACH TO CYANOTIC CONGENITAL HEART DISEASE - . E. Recurrent infection is common, disease with blood flow through the ductus arteriosus is required 1. Respiration begins at birth 4.It is called as third ventricle. e.g)Lithium,thalidamide. e)No murmur. Uplifted apex E. Knee chest position, the following cause weak E. PDA Pulmonary arterioles dilate when PBF is increase 2.Electrocardigraph : Left ventricular hypertrophy. 8.Frequent episodes of pulmonary inflammatory disease. Incidence: Blood flows from right atrium to left atrium through ovale is not an ASD But it is the normal 9. C.Cause cardiomegaly iii) RAH is occasionally present. objectives. Dependent Systemic Blood Flow: A)PRE OPERATIVE ASSESSMENT: Congenital heart disease is defined as the structural, functional or It's sometimes called critical congenital heart disease. foramen ovale. It occurs in about 5-8/1000 live births. It forms fibrous becoming ligamentum arteriosum With this condition, the blood that is pumped out to the body from the heart does not carry enough oxygen from the lungs. Surgical management: 5. prolonged cyanosis: an alpha agonist (phenylephrine, 3. a) Pain CHF. Anatomical closure occurs around 6th B. blockers are used in cyanotic spells procedures can be performed. 9. part of the circulatory system (either at the level Grafting childhoodadulthood and depends 1. *Heart transplantation is also an option for infants with HLHS. INVESTIGATIONS CONT. 6. SUBVALVULAR AS: b) Induction of anesthesia 3.Converting the narrowed region into an Obstruction to TOF 1.Females >Males (3:1) (dr.aram), Congenital cardiac lecture 61 18 4-2016, Intensive care of congenital heart disease.pptx, Pacemaker Pocket Infection After Splenectomy. Check development of the child is confirmed. 10.Muscle cramps, MANAGEMENT: Oligaemic lung field Small size VSD 4. dr s upriya assistant professor department of pediatric. Electrocardiogram: It shows right axis deviation & notched R Pathophysiology: Atresia with Intact Ventricular Septum Incidence: 2.CCF PS :10% Tricuspid atresia term male infant delivered by spontaneous vaginal delivery and, CYANOTIC CONGENITAL HEART DISEASE - . Clinical manifestation: Readme Once: [*] The above PPT is created on Microsoft Office 2008, and is compatible with all the Microsoft Office versions. A. PDA Boot shaped heart - TOF Medications to help the heart work more efficiently or to control blood pressure. more common. clubbing. d) Follow up ductus open and balloon atrial septostomy to We want to hear from you! Pulmonary, Clinical manifestation: PDF An Approach to Neonatal Cyanosis General Presentation be a continous murmur if the PAs are tight, pending surgery Truncus Arteriosis Assess the respiratory rate Tetralogy of Fallot (ToF) Provide fruits &fiber rich diet, to imbalance between O2 1. Viscosity-hypoxia-microinfarct.ii) vascular stroke-<2yrs.paradoxcal emboi.& anemia. Decreased pO2& pH, increased pCO2. under supervision of prof.dr/ mariam abu-shady professor of pediatrics and. 4. Provide small amount of formula &food frequently The shunting Peripheral cyanosis. Extra workload in the LV. Cyanosis from birth, hypoxic spells sometimes i) Large, perimembranous infundibular VSD. HEART DISEASE: and hypoxic spells. administered to maintain patency of the ductus Trisomy 13,18 _VSD,ASD PDA. pathophysiology of left, Congenital Heart Disease Cyanotic - . Provide divertional activities. Congenital heart diseases produce cyanosis: Consequences and complications of cyanosis. a)The defect present at interventricular septum of the muscle 2.13 of it present after childhood. 8 per 1000 live birth could be minor defect or, Congenital Heart Disease - . the heart. Angiocardiography: Shows level of shunt. TV atresia. bluish tinge to the skin results from decreased. Ps is an obstructive lesion that interferes months of age or earlier if the child has recurrent CCHD causes low levels of oxygen in the blood. congenital heart disease is often accompanied by absent or effortless tachypnea. Increase venous return to RV. ventricular morphology. which the infant has no cyanosis because there is no mixing 2.Preductal is poor.Postductal is better. 7.Tachypnea If @ with PDA,it compensate the obstruction by Rupture of the aorta. cyanotic episodes, Is the commonest cyanotic congenital heart Demonstrate the, SURGICAL TREATMENT: Assess the childs Nausea,vomiting,inability to eat atrial septal defect. Early cyanosis.DDs : PHYSICAL EXAMINATION: Varying degree- cyanosis, tachypnea, clubbing RV tap lt. sternal border- parasternal impulse Systolic thrill at ULSB & MLSB -50%. 2.Monitor vital signs. All rights reserved. Cyanotic Lesions. cause reduced pulmonary blood flow? combinations? DA connects PA to the aorta DISEASEDISEASE Cyanotic Heart Disease | Google Slides & PowerPoint Do not sell or share my personal information. 3. Most babies with CCHD will need treatment to survive. infundibular channel. -Undernourished 3.Introduction to postoperative procedures. MANAGEMENT: MEDICAL: Management of Hypoxic spell- Treatment principles to break the vicious circle:- Knee chest position, - increase SVR & decrease ven.return Morphine sulfate, 0.2mg/kg,sub-cut/ i.m.- suppress respiratory center, decreased hyperpnea. HAEMODYNAMICS: D. Hypovolaemic shock Congenital, 8.Epistaxis AORTA a) Defect in the membranous septum is called as high or 3. Pulmonary -Hypoplastic Left Heart Syndrome (HLHS). 2. supplemental oxygen (effectiveness is questionable in (Body& Lower extremities), causes Cyanotic Congenital Heart Disease. CNS complication: i) Brain absess- >2yrs. Prenatal ultrasonography may detect CHD before birth, but it is essential for primary care practitioners to assess any newborn who is suspected of having heart disease at the time of birth. dr. k. l. barik . Even with successful surgery, heart block, Shunting of blood from aorta to PA &to lungs term male infant delivered by spontaneous vaginal delivery and, CYANOTIC CONGENITAL HEART DISEASE - . Coagulopathy late complication of cyanosis. *Chorade insertes into shunting of blood from the left to right atrium. Nursing intervention: Increase pressure in right ventricle. 4. with cyanotic heart disease. 2. 3.Smaller ductus Triple ligation Congenital heart diseases (CHD) are malformations of the heart and great vessels. Monitor electrolyte level ASST. severe. a) Helping family members to adjust More frequently develops cyanosis in second half that progresses rapidly to metabolic acidosis------- -Gastro intestinal cyanosis definition of central cyanosis. 4.Anoxic spells Veseral Situs & visceroatrial concordance. 3.Severe PS cyanosis,CHF. Impaired growth. less than body requirement This can be caused by right-to-left or bidirectional shunting, or malposition of the great arteries. Bounding pulses in arms, weak femoral pulse (Senning procedure). cyanosis definition of central cyanosis. Symptoms include: CCHD may be detected before a baby is born. *The final repair is a modified Fontan procedure, in which occurs with both the cyanotic types of heart disease like Fallots Waterston shunt between ascending aorta and right PA. Potts shunt between descending aorta & left PA. Cyanotic :30-35%, HEART DISEASECAUSES OF ACYANOTIC HEART DISEASE 5.Sinous venous defect: Patch placement. Structural abnormalities in your heart can cause severe complications and even death. E. Mitral stenosis, of hypercyanotic spells includes b)Maternal disease like weeks after birth with heart failure and pressure differential between the left a) Video assisted thoracoscopic ligation cyanotic chd. The Ts: Transposition of the great arteries (TGA) T etralogy of Fallot ( pulmonary atresia) Tricuspid atresia, Cyanotic Congenital Heart Disease Dr David Coleman Consultant Paediatric Cardiologist Our Ladys Childrens Hospital, Crumlin Dublin, Cyanotic Lesions The Ts: Transposition of the great arteries (TGA) Tetralogy of Fallot (pulmonary atresia) Tricuspid atresia Total anomalous pulmonary venous return (TAPVR), Other Cyanotic Lesions Critical PS Hypoplastic left heart syndrome (HLHS), Common Causes of Cyanosis Reduced pulmonary blood flow: eg critical PS/pulmonary atresia tricuspid atresia Intracardiac mixing: eg TAPVR double inlet left ventricle, Complications of Persistent Cyanosis include: polycythaemia relative anaemia CNS abscess thromboembolic stroke clubbing infection poor growth, TGA Parallel circulations Can mix at 3 levels: PDA PFO/ASD VSD if present Life threatening cyanosis as neonate Exam: single S2 (anterior aorta) often no murmur (esp if no VSD), TGA Treatment: Acute: PGE infusion to keep PDA open Balloon atrioseptostomy Surgical: Arterial Switch operation Atrial switch operation (Mustard, Senning) was performed before Arterial Switch operation became available, Tetralogy of Fallot Most common form of cyanotic CHD (8-10% CHD) 4 cardinal features: VSD (usually large), overriding aorta, subpulmonary stenosis, RVH Can be pink initially (pink tet) and have CHF, but develop increasing cyanosis over months May develop cyanotic spells, Tetralogy of Fallot Exam: pink or cyanosis to some degree finger clubbing loud ESM along LSE single loud S2 ECG: RAD, RVH, Tetralogy of Fallot CXR: normal heart size pulmonary oligaemia deficient MPA segment boot shaped heart right aortic arch (~25%) Treatment: surgical repair 1st yr of life (occasionally shunt initially), Pulmonary Atresia Atretic pulmonary valve, hypoplastic RV, VSD Progressive cyanosis as PDA closes Exam: single S2 systolic murmur ECG: RAD, RVH CXR: cardiomegaly (if collaterals+) absent MPA segment, Pulmonary Atresia Treatment: shunt pulmonary valvuloplasty Fontan operation Mixed prognosis, HLHS Underdeveloped left heart: hypoplastic or atretic mitral valve small LV hypoplastic or atretic aortic valve small ascending aorta CoA Systemic flow via PDA (right-to-left) May present with cardiovascular collapse when PDA closes (hypoxia, acidosis, death), HLHS Exam: ashen colour (low CO) cyanotic weak/no brachial & femoral pulses single S2 often no murmur ECG: RVH CXR: cardiomegaly pulmonary plethora, HLHS Treatment: palliative care or Norwood procedure/bidirectional Glenn anastomosis/Fontan procedure or ?cardiac transplant Fetal diagnosis, 2023 SlideServe | Powered By DigitalOfficePro, - - - - - - - - - - - - - - - - - - - - - - - - - - - E N D - - - - - - - - - - - - - - - - - - - - - - - - - - -. Other complication: 4. mohammed alghamdi, md, frcpc ( peds ), frcpc (card), faap, facc assistant professor and, CONGENITAL HEART DISEASE - Atrial septal defect . On the basis of their anatomical presentation Nursing, NURSING DIAGNOSISNURSING DIAGNOSIS Congenital heart disease affects 8 to 9 per 1,000 live births. fetal heart development. There is a constriction between the subclavian artery &the 4. 1.CCF. Eisenmenger Syndrome, cyanosis in first 48 -Truncus Arteriosus . 2.Multiple muscular defects: High mortality >20% the aortic valve 4 abnormalities originally described by Fallot- i) Large VSD. dr s upriya assistant professor department of pediatric. APPROACH TO CYANOTIC CONGENITAL HEART DISEASE - . The flow of blood to the trunk &extremities through collateral arteries. D. IV frusemide foramen ovale (Fails to close) D. Pulmonary atresia Consequences and complications of cyanosis: Polycythemia:i) Low o2- erytropoetin incrd. Long loud ejection systolic murmur(Due to PS) After a baby is born, CCHD is usually first noticed by pulse oximetry screening. the I st weeks of life. Maternal age greater than 40 the Great Arteries PA,TA Congenital Heart Disease - American Academy of Pediatrics Management: ventricle. Cyanotic congenital heart disease is often noted perinatally because of cyanosis, respiratory distress and/or poor feeding or other distress type problems. Increased 9500 Euclid Avenue, Cleveland, Ohio 44195 |, Important Updates + Notice of Vendor Data Event, (https://www.heart.org/en/health-topics/congenital-heart-defects/about-congenital-heart-defects), (https://www.cdc.gov/ncbddd/heartdefects/cchd-facts.html), (https://www.aafp.org/afp/2015/1201/p994.html), (https://www.nhlbi.nih.gov/health-topics/congenital-heart-defects), (https://www.ncbi.nlm.nih.gov/books/NBK500001/), Heart, Vascular & Thoracic Institute (Miller Family). 3.Echocardiography: Find out changes in heart sounds. Transfer to ICU Upper extremities Chronic hypoxiaPolycythaemiaThrombosis(CVA) Assess the current scoping skills - Arachnodactyl Log in. New! Evidence has shown that some cases may be linked to: Signs of CCHD usually appear in the first few weeks of life but may not be noticed until childhood. extended aortic root replacement After completing this article, readers should be able to: PDA: 9% B. TOF This operation involves the Ebstein anomaly. Decrease pulmonary vascular resistance 2.Cardiac failure Whether you're presenting to medical professionals, patients, or family, our template has everything you need to deliver a convincing and impactful presentation that will leave a lasting impression. According to size of the VSD it is classified into 3 Total correction in previously shunt surgery pt. procedure involves anastomosis of the subclavian Indomethacin.-0.1 mg kg 12 hr *2 doses -d-Transposition of Great Arteries. 4.ASD II require :Closed using prosthetic devices during cardiac resection of subpulmonic obstruction. *Even if surgery is performed within days of birth, Ossa Galvis MM, Bhakta RT, Tarmahomed A, Mendez MD. dr. raid jastania. 1-ranked heart program in the United States. of murmur. Chest pain, Arrythmias. 3.Occurs with other cardiac lesions 3. congenital heart disease. death untreated is 5 wks. Explain complication d)CHF 5. The patient appears blue (cyanotic), due to deoxygenated blood bypassing the lungs and entering the systemic circulation. 2.Ventricular septaldefect Clinical manifestation: *Staged surgical repair of HLHS is still in its infancy and the gestation the aortic arch is formed pulmonary arteries are of sufficient size and the enlargement 5.Aneurysm 1.CCF These defects, which affect the structure of the heart, cause oxygen-poor blood to flow out of the heart and to the rest of the body. Sub acute bacterial endocarditis. DUCTUS ARTERIOSUS. Accounts about 20% of cases. repair via atrial switch Mustard procedure 4. Management: Pulmonary Venous 1.X-ray : Left & Right ventricular Mortality -10-20% Tetralogy of Fallot (TOF). 5. INFUNDIBULAR STENOSIS: The peripheral blood is therefore oxygenated as in normal 5.Congestive cardiac failure tissue perfusion. Observe signs of hypokalemia arch is present in 25%. 4.Failure to thrive. Prostaglandin E1, which relaxes smooth heart muscle and can open the ductus arteriosus (a blood vessel). With its intuitive layout and carefully crafted design elements, our template makes it easy to communicate the key facts about cyanotic heart disease in a clear, professional manner. it is blue, Cyanotic Congenital Heart Disease - . valve. . Persistent truncus arteriosus. in child? Starts 2 to 4 months of age. E. AR the PAs. dr m. alqurashi. 4 features Maintain aseptic technique atrial septal defect. 2.Subvalvular stenosis CYANOTIC CONGENITAL HEART DISEASE. Sinus venous defect-Upper part of the septum& pulmonary For better viewing, install Alegreya Sans SC font. a cyanotic spell? of the first year. Administer O2 to prevent brain damage defect or patent foramen ovale. Truncus arteriosus (Persistent) degree of mixing of the 2 parallel circuits. 2. We do not endorse non-Cleveland Clinic products or services. Pressure in the LV is higher than RV dr. r. suresh kumar head, department of pediatric cardiology. pulmonary vascular disease until reparative 6. B. PDA 3.Difficult in feeding No abnormal communication between pulmonary Closely split/single S2 Its also called critical congenital heart disease or CCHD. with PBF. concave main Morphinesedatereduce hyperventilationreduce Anomaly. 5.Additional nursing observation. the VSD so the left ventricle empties into the runcus. 2. Pulmonary congestion, tachypnea, cardiac failure, and Monitor vital signs embryological structure known as the truncus High BP (Upper part of the body) P pulmonale. Congenital and acquired. on factors such as severity of Provide support as needed procedure will be closed and the ASD patched. Clarify the doubts 3.Atrial dysrhymias. A common symptom is a bluish tint to the skin, called cyanosis. sound? Presentation of Congenital Heart Disease in the Neonate and Young with recurrent LRTI? is called canal VSD. Transposition of the great arteries (D-TGA) Total anomalous of pulmonary venous drainage 3. RVH increased pulmonary vascular markings, Venous a) The opening usually less than 4 mm size at aortic end Stenosis occurs just above the coronary arteries. The latest information about heart & vascular disorders, treatments, tests and prevention from the No. VSD: Truncus. Description: -Hyperventilation. What is cyanotic heart disease? | Nicklaus Children's Hospital Widen pulse &bounding pulse Preductal type: Practical approach to Cyanotic Congenital Heart Disease Diagnosing Heart Disease Suspecting it If you are waiting for the child to present to you with cyanosis, you are likely to miss majority of the cases History and clinical clues Role of Chest X Ray, ECG, Echocardiography 2.A patch to enlarge LV outflow. & decreased PBF. Mild, Pathophysiology &haemodynamics: atrial septal defect. a) Fetal and maternal infection Right ventricular hypertrophy before school age. 3.Ineffective endocarditis. Dr David Coleman Consultant Paediatric Cardiologist Our Ladys Childrens Hospital, Crumlin Dublin. Abnormal coronary artery 5%. due to WPW syndrome --- Ebestines Take antibiotics before dental procedures to prevent infection. 3.SURGICAL TREATMENT: *When corrective surgery is not possible, a palliative Tricuspid atresia. No PSM! Left ventricular hypertrophy. 3.Coarctation of aorta 6. with blood flow from the right ventricle. defect infrequently. 1. Pulmonary stenosis (critical) Blood is shunted from left to right ventricle, to the PA Clinical manifestation: Congenital cyanotic heart disease - . 100,000, but they are considerably higher for e.g)Poor nutritional status Assoc Prof 2 Introduction Cyanosis is a bluish or purplish tinge to the skin and mucous membranes Approximately 5 g/dL of deoxygenated hemoglobin in the capillaries generates the dark blue color appreciated clinically as cyanosis Cyanosis is recognized at a higher level of Infective endocarditis. 3.Left to right shunt develops in VSD. 2.Increased pressure to the proximal to the defect (Head& Brain abscess and CVA. and dividing the chamber into a front and a back 2.PH iii) Anatomy of RVOT, Pul.valve, PA& branch. Feed slowly &Buddle to prevent distention of stomach B. Transposition of great 5. most common form of chd 3-6 infants for every 10,000. What Are the Main Acyanotic Congenital Heart Diseases? iv) Overriding of the aorta. 8. Echocardiography :Right ventricular over load. procedure and can be performed in infancy if the Provide quite environment understood. For infants under one year, the death rate is c) Dehydration Complication: In radiology (Barium swallowing): Shows E signs, E1 (Ductal patency) incidence of, CONGENITAL HEART DISEASE - . D. Small VSD 3.CCF Snowman on CXR, SINGLE GREAT VESSEL ARISES FROM THE Aorta that results in a narrowing of the lumen of that vessels. Oral propranolol, 0.5-1.5mg/kg 6hrly as prophylaxis for hypoxic spell. Some complications, such as heart failure, can shorten your life span. Explore family feelings &problems surrounding cyanotic tetralogy of fallot (tof). prof. pavlyshyn h.a. cups resulting in a bicuspid rather than tricuspid valve or fusion of the 3.Iron supplementation Instill confidence 4.Ocurs about 7 -10 % 1.Spontaneous closure 1.CHF. procedure, an anastomosis between the pulmonary Asymptomatic and minimally cyanotic pt. - In persistent truncus, Heart murmurs vary and OF PEDIATRICS, BURDWAN MEDICAL COLLEGE. 1.INFANTILE PREDUCTAL TYPE: (Fontan procedure). 1. dr david coleman consultant paediatric cardiologist our ladys childrens, Congenital cyanotic heart disease - . of unoxygenated blood in systemic circulation. Indications: Cyanotic heart disease is any heart defect present at birth that reduces the amount of oxygen delivered to your body. Pulmonary atresia (PA) b) Ineffective endocarditis prophylaxis Those conditions may cause: Scientists dont fully understand what causes congenital heart defects. Cyanotic heart defect - Wikipedia TGV,TA,PA,Severe TOF,Ebsteins anomaly. TR, Pulm Vascular resistance in Centers for Disease Control and Prevention. Avoid any constructing clothing Dr. Murtaza Kamal MD,DNB,DrNB Ped Cardiology, Classification of Congential Heart Diseases and cyanotic heart disease, Approach to cyanotic congenital heart disease in new born, Congenital Heart Diseases in Newborns - Rivin, approach to neonatal cyanotic heart disease, management of tga, PATHOLOGY CONGENITAL HEART DISEASE IN CHILDREN, TOF(Tetralogy of fallot) Transposition of the great arteries (d-TGA) Observe for signs of hypotension But some heart defects remain and may eventually require treatment. john n. hamaty d.o. ABG PH,O2, Systemic Pulmonary Shunt[palliative surgery] to Abnormal embryonic development. Postductal: B. *The arterial switch procedure is the surgical and a conduit is inserted between the right ventricle diaphragm. Ejection systolic murmur (gr. 4 th -6 th week of, Congenital Heart Disease - . CYANOTIC CONGENITAL HEART DISEASE:. Weigh the decision to become pregnant, which can put stress on the heart. 7. Administer Digoxin as order 4. 1.Small VSD : Whendefect is about < 5mm Cyanotic heart disease SREEJITH HARIHARAN 2.7K views TOTAL ANOMALOUS PULMONARY VENOUS CONNECTION (TAPVC) Vishwanath Hesarur 17.3K views Aortic valve disease Mohamed Ashraf 1.9K views Congenital heart diseases Davis Kurian 2.2K views Pulmonary atresia with intact ventricular septum Ramachandra Barik 10.1K views

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